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25th Annual Computational Neuroscience Meeting: CNS-2016.

BMC neuroscience

[No authors listed]
PMID: 27534393
BMC Neurosci. 2016 Aug 18;17:54. doi: 10.1186/s12868-016-0283-6.

No abstract available.

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25th Annual Computational Neuroscience Meeting: CNS-2016. BMC Neurosci. 2016;17:54doi: 10.1186/s12868-016-0283-6.
(2016). 25th Annual Computational Neuroscience Meeting: CNS-2016. BMC neuroscience, 1754. https://doi.org/10.1186/s12868-016-0283-6
"25th Annual Computational Neuroscience Meeting: CNS-2016." BMC neuroscience vol. 17 (2016): 54. doi: https://doi.org/10.1186/s12868-016-0283-6
25th Annual Computational Neuroscience Meeting: CNS-2016. BMC Neurosci. 2016 Aug 18;17:54. doi: 10.1186/s12868-016-0283-6. PMID: 27534393; PMCID: PMC5001212.
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Spinal reflexive movement follows general tau theory.

BMC neuroscience

Bahadori M, Cesari P, Craig C, Andani ME.
PMID: 33794775
BMC Neurosci. 2021 Apr 01;22(1):23. doi: 10.1186/s12868-021-00626-3.

BACKGROUND: Tau theory explains how both intrinsically and perceptually guided movements are controlled by the brain. According to general tau theory, voluntary, self-paced human movements are controlled by coupling the tau of the movement (i.e., the rate of closure...

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Bahadori M, Cesari P, Craig C, et al. Spinal reflexive movement follows general tau theory. BMC Neurosci. 2021;22(1):23doi: 10.1186/s12868-021-00626-3.
Bahadori, M., Cesari, P., Craig, C., & Andani, M. E. (2021). Spinal reflexive movement follows general tau theory. BMC neuroscience, 22(1), 23. https://doi.org/10.1186/s12868-021-00626-3
Bahadori, Mehrdad, et al. "Spinal reflexive movement follows general tau theory." BMC neuroscience vol. 22,1 (2021): 23. doi: https://doi.org/10.1186/s12868-021-00626-3
Bahadori M, Cesari P, Craig C, Andani ME. Spinal reflexive movement follows general tau theory. BMC Neurosci. 2021 Apr 01;22(1):23. doi: 10.1186/s12868-021-00626-3. PMID: 33794775; PMCID: PMC8015145.
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Differential effects of rat ADSCs encapsulation in fibrin matrix and combination delivery of BDNF and Gold nanoparticles on peripheral nerve regeneration.

BMC neuroscience

Razavi S, Jahromi M, Vatankhah E, Seyedebrahimi R.
PMID: 34384370
BMC Neurosci. 2021 Aug 12;22(1):50. doi: 10.1186/s12868-021-00655-y.

BACKGROUND: Fibrin as an extracellular matrix feature like biocompatibility, creates a favorable environment for proliferation and migration of cells and it can act as a reservoir for storage and release of growth factors in tissue engineering.METHODS: In this study,...

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Razavi S, Jahromi M, Vatankhah E, et al. Differential effects of rat ADSCs encapsulation in fibrin matrix and combination delivery of BDNF and Gold nanoparticles on peripheral nerve regeneration. BMC Neurosci. 2021;22(1):50doi: 10.1186/s12868-021-00655-y.
Razavi, S., Jahromi, M., Vatankhah, E., & Seyedebrahimi, R. (2021). Differential effects of rat ADSCs encapsulation in fibrin matrix and combination delivery of BDNF and Gold nanoparticles on peripheral nerve regeneration. BMC neuroscience, 22(1), 50. https://doi.org/10.1186/s12868-021-00655-y
Razavi, Shahnaz, et al. "Differential effects of rat ADSCs encapsulation in fibrin matrix and combination delivery of BDNF and Gold nanoparticles on peripheral nerve regeneration." BMC neuroscience vol. 22,1 (2021): 50. doi: https://doi.org/10.1186/s12868-021-00655-y
Razavi S, Jahromi M, Vatankhah E, Seyedebrahimi R. Differential effects of rat ADSCs encapsulation in fibrin matrix and combination delivery of BDNF and Gold nanoparticles on peripheral nerve regeneration. BMC Neurosci. 2021 Aug 12;22(1):50. doi: 10.1186/s12868-021-00655-y. PMID: 34384370; PMCID: PMC8359623.
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Eosinophil-to-monocyte ratio is a potential biomarker in the prediction of functional outcome among patients with acute ischemic stroke.

BMC neuroscience

Yu S, Luo Y, Zhang T, Huang C, Fu Y, Zhang Q, Zeng F, Huang H, Zhang C, Guo Z.
PMID: 33546596
BMC Neurosci. 2021 Feb 05;22(1):8. doi: 10.1186/s12868-021-00610-x.

BACKGROUND: It has been shown that eosinophils are decreased and monocytes are elevated in patients with acute ischemic stroke (AIS), but the impact of eosinophil-to-monocyte ratio (EMR) on clinical outcomes among AIS patients remains unclear. We aimed to determine...

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Yu S, Luo Y, Zhang T, et al. Eosinophil-to-monocyte ratio is a potential biomarker in the prediction of functional outcome among patients with acute ischemic stroke. BMC Neurosci. 2021;22(1):8doi: 10.1186/s12868-021-00610-x.
Yu, S., Luo, Y., Zhang, T., Huang, C., Fu, Y., Zhang, Q., Zeng, F., Huang, H., Zhang, C., & Guo, Z. (2021). Eosinophil-to-monocyte ratio is a potential biomarker in the prediction of functional outcome among patients with acute ischemic stroke. BMC neuroscience, 22(1), 8. https://doi.org/10.1186/s12868-021-00610-x
Yu, Shuhong, et al. "Eosinophil-to-monocyte ratio is a potential biomarker in the prediction of functional outcome among patients with acute ischemic stroke." BMC neuroscience vol. 22,1 (2021): 8. doi: https://doi.org/10.1186/s12868-021-00610-x
Yu S, Luo Y, Zhang T, Huang C, Fu Y, Zhang Q, Zeng F, Huang H, Zhang C, Guo Z. Eosinophil-to-monocyte ratio is a potential biomarker in the prediction of functional outcome among patients with acute ischemic stroke. BMC Neurosci. 2021 Feb 05;22(1):8. doi: 10.1186/s12868-021-00610-x. PMID: 33546596; PMCID: PMC7863468.
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Correction to: Eye-tracking-aided characterization of saccades and antisaccades in SYNE1 ataxia patients: a pilot study.

BMC neuroscience

Szpisjak L, Szaraz G, Salamon A, Nemeth VL, Szepfalusi N, Veres G, Kincses B, Maroti Z, Kalmar T, Rydzanicz M, Ploski R, Klivenyi P, Zadori D.
PMID: 33691616
BMC Neurosci. 2021 Mar 10;22(1):16. doi: 10.1186/s12868-021-00621-8.

No abstract available.

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Szpisjak L, Szaraz G, Salamon A, et al. Correction to: Eye-tracking-aided characterization of saccades and antisaccades in SYNE1 ataxia patients: a pilot study. BMC Neurosci. 2021;22(1):16doi: 10.1186/s12868-021-00621-8.
Szpisjak, L., Szaraz, G., Salamon, A., Nemeth, V. L., Szepfalusi, N., Veres, G., Kincses, B., Maroti, Z., Kalmar, T., Rydzanicz, M., Ploski, R., Klivenyi, P., & Zadori, D. (2021). Correction to: Eye-tracking-aided characterization of saccades and antisaccades in SYNE1 ataxia patients: a pilot study. BMC neuroscience, 22(1), 16. https://doi.org/10.1186/s12868-021-00621-8
Szpisjak, Laszlo, et al. "Correction to: Eye-tracking-aided characterization of saccades and antisaccades in SYNE1 ataxia patients: a pilot study." BMC neuroscience vol. 22,1 (2021): 16. doi: https://doi.org/10.1186/s12868-021-00621-8
Szpisjak L, Szaraz G, Salamon A, Nemeth VL, Szepfalusi N, Veres G, Kincses B, Maroti Z, Kalmar T, Rydzanicz M, Ploski R, Klivenyi P, Zadori D. Correction to: Eye-tracking-aided characterization of saccades and antisaccades in SYNE1 ataxia patients: a pilot study. BMC Neurosci. 2021 Mar 10;22(1):16. doi: 10.1186/s12868-021-00621-8. PMID: 33691616; PMCID: PMC7944885.
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Eye movement characteristics in male patients with deficit and non-deficit schizophrenia and their relationships with psychiatric symptoms and cognitive function.

BMC neuroscience

Zhang L, Zhang X, Fang X, Zhou C, Wen L, Pan X, Zhang F, Chen J.
PMID: 34819034
BMC Neurosci. 2021 Nov 24;22(1):70. doi: 10.1186/s12868-021-00673-w.

BACKGROUND: The cognitive impairment pattern of deficit schizophrenia (DS) is centered on an impaired attention function. Previous studies have suggested that the exploratory eye movement (EEM) tests reflect attention deficits in patients with schizophrenia. However, no study has investigated...

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Zhang L, Zhang X, Fang X, et al. Eye movement characteristics in male patients with deficit and non-deficit schizophrenia and their relationships with psychiatric symptoms and cognitive function. BMC Neurosci. 2021;22(1):70doi: 10.1186/s12868-021-00673-w.
Zhang, L., Zhang, X., Fang, X., Zhou, C., Wen, L., Pan, X., Zhang, F., & Chen, J. (2021). Eye movement characteristics in male patients with deficit and non-deficit schizophrenia and their relationships with psychiatric symptoms and cognitive function. BMC neuroscience, 22(1), 70. https://doi.org/10.1186/s12868-021-00673-w
Zhang, Lin, et al. "Eye movement characteristics in male patients with deficit and non-deficit schizophrenia and their relationships with psychiatric symptoms and cognitive function." BMC neuroscience vol. 22,1 (2021): 70. doi: https://doi.org/10.1186/s12868-021-00673-w
Zhang L, Zhang X, Fang X, Zhou C, Wen L, Pan X, Zhang F, Chen J. Eye movement characteristics in male patients with deficit and non-deficit schizophrenia and their relationships with psychiatric symptoms and cognitive function. BMC Neurosci. 2021 Nov 24;22(1):70. doi: 10.1186/s12868-021-00673-w. PMID: 34819034; PMCID: PMC8613938.
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Retraction Note to: miR-320a affects spinal cord edema through negatively regulating aquaporin-1 of blood-spinal cord barrier during bimodal stage after ischemia reperfusion injury in rats.

BMC neuroscience

Li XQ, Fang B, Tan WF, Wang ZL, Sun XJ, Zhang ZL, Ma H.
PMID: 34732132
BMC Neurosci. 2021 Nov 03;22(1):64. doi: 10.1186/s12868-021-00669-6.

No abstract available.

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Li XQ, Fang B, Tan WF, et al. Retraction Note to: miR-320a affects spinal cord edema through negatively regulating aquaporin-1 of blood-spinal cord barrier during bimodal stage after ischemia reperfusion injury in rats. BMC Neurosci. 2021;22(1):64doi: 10.1186/s12868-021-00669-6.
Li, X. Q., Fang, B., Tan, W. F., Wang, Z. L., Sun, X. J., Zhang, Z. L., & Ma, H. (2021). Retraction Note to: miR-320a affects spinal cord edema through negatively regulating aquaporin-1 of blood-spinal cord barrier during bimodal stage after ischemia reperfusion injury in rats. BMC neuroscience, 22(1), 64. https://doi.org/10.1186/s12868-021-00669-6
Li, Xiao-Qian, et al. "Retraction Note to: miR-320a affects spinal cord edema through negatively regulating aquaporin-1 of blood-spinal cord barrier during bimodal stage after ischemia reperfusion injury in rats." BMC neuroscience vol. 22,1 (2021): 64. doi: https://doi.org/10.1186/s12868-021-00669-6
Li XQ, Fang B, Tan WF, Wang ZL, Sun XJ, Zhang ZL, Ma H. Retraction Note to: miR-320a affects spinal cord edema through negatively regulating aquaporin-1 of blood-spinal cord barrier during bimodal stage after ischemia reperfusion injury in rats. BMC Neurosci. 2021 Nov 03;22(1):64. doi: 10.1186/s12868-021-00669-6. PMID: 34732132; PMCID: PMC8565068.
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Post-status epilepticus treatment with the Fyn inhibitor, saracatinib, improves cognitive function in mice.

BMC neuroscience

Luo XM, Zhao J, Wu WY, Fu J, Li ZY, Zhang M, Lu J.
PMID: 33451301
BMC Neurosci. 2021 Jan 15;22(1):2. doi: 10.1186/s12868-020-00606-z.

BACKGROUND: Status epilepticus (SE) is a life-threatening neurological disorder. The hippocampus, as an important area of the brain that regulates cognitive function, is usually damaged after SE, and cognitive deficits often result from hippocampal neurons lost after SE. Fyn,...

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Luo XM, Zhao J, Wu WY, et al. Post-status epilepticus treatment with the Fyn inhibitor, saracatinib, improves cognitive function in mice. BMC Neurosci. 2021;22(1):2doi: 10.1186/s12868-020-00606-z.
Luo, X. M., Zhao, J., Wu, W. Y., Fu, J., Li, Z. Y., Zhang, M., & Lu, J. (2021). Post-status epilepticus treatment with the Fyn inhibitor, saracatinib, improves cognitive function in mice. BMC neuroscience, 22(1), 2. https://doi.org/10.1186/s12868-020-00606-z
Luo, Xin-Ming, et al. "Post-status epilepticus treatment with the Fyn inhibitor, saracatinib, improves cognitive function in mice." BMC neuroscience vol. 22,1 (2021): 2. doi: https://doi.org/10.1186/s12868-020-00606-z
Luo XM, Zhao J, Wu WY, Fu J, Li ZY, Zhang M, Lu J. Post-status epilepticus treatment with the Fyn inhibitor, saracatinib, improves cognitive function in mice. BMC Neurosci. 2021 Jan 15;22(1):2. doi: 10.1186/s12868-020-00606-z. PMID: 33451301; PMCID: PMC7811255.
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The inflammatory profile of cerebrospinal fluid, plasma, and saliva from patients with severe neuropathic pain and healthy controls-a pilot study.

BMC neuroscience

Jönsson M, Gerdle B, Ghafouri B, Bäckryd E.
PMID: 33522900
BMC Neurosci. 2021 Feb 01;22(1):6. doi: 10.1186/s12868-021-00608-5.

BACKGROUND: Neuropathic pain (NeuP) is a complex, debilitating condition of the somatosensory system, where dysregulation between pro- and anti-inflammatory cytokines and chemokines are believed to play a pivotal role. As of date, there is no ubiquitously accepted diagnostic test...

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Jönsson M, Gerdle B, Ghafouri B, et al. The inflammatory profile of cerebrospinal fluid, plasma, and saliva from patients with severe neuropathic pain and healthy controls-a pilot study. BMC Neurosci. 2021;22(1):6doi: 10.1186/s12868-021-00608-5.
Jönsson, M., Gerdle, B., Ghafouri, B., & Bäckryd, E. (2021). The inflammatory profile of cerebrospinal fluid, plasma, and saliva from patients with severe neuropathic pain and healthy controls-a pilot study. BMC neuroscience, 22(1), 6. https://doi.org/10.1186/s12868-021-00608-5
Jönsson, Mika, et al. "The inflammatory profile of cerebrospinal fluid, plasma, and saliva from patients with severe neuropathic pain and healthy controls-a pilot study." BMC neuroscience vol. 22,1 (2021): 6. doi: https://doi.org/10.1186/s12868-021-00608-5
Jönsson M, Gerdle B, Ghafouri B, Bäckryd E. The inflammatory profile of cerebrospinal fluid, plasma, and saliva from patients with severe neuropathic pain and healthy controls-a pilot study. BMC Neurosci. 2021 Feb 01;22(1):6. doi: 10.1186/s12868-021-00608-5. PMID: 33522900; PMCID: PMC7852144.
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Metformin attenuates sepsis-induced neuronal injury and cognitive impairment.

BMC neuroscience

Qin Z, Zhou C, Xiao X, Guo C.
PMID: 34911449
BMC Neurosci. 2021 Dec 15;22(1):78. doi: 10.1186/s12868-021-00683-8.

BACKGROUND: Sepsis is considered to be a high-risk factor for cognitive impairment in the brain. The purpose of our study is to explore whether sepsis causes cognitive impairment and try to evaluate the underlying mechanisms and intervention measures.METHODS: Here,...

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Qin Z, Zhou C, Xiao X, et al. Metformin attenuates sepsis-induced neuronal injury and cognitive impairment. BMC Neurosci. 2021;22(1):78doi: 10.1186/s12868-021-00683-8.
Qin, Z., Zhou, C., Xiao, X., & Guo, C. (2021). Metformin attenuates sepsis-induced neuronal injury and cognitive impairment. BMC neuroscience, 22(1), 78. https://doi.org/10.1186/s12868-021-00683-8
Qin, Zhenghui, et al. "Metformin attenuates sepsis-induced neuronal injury and cognitive impairment." BMC neuroscience vol. 22,1 (2021): 78. doi: https://doi.org/10.1186/s12868-021-00683-8
Qin Z, Zhou C, Xiao X, Guo C. Metformin attenuates sepsis-induced neuronal injury and cognitive impairment. BMC Neurosci. 2021 Dec 15;22(1):78. doi: 10.1186/s12868-021-00683-8. PMID: 34911449.
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Correction to: Wnt3a induces exosome secretion from primary cultured rat microglia.

BMC neuroscience

Hooper C, Sainz-Fuertes R, Lynham S, Hye A, Killick R, Warley A, Bolondi C, Pocock J, Lovestone S.
PMID: 32138650
BMC Neurosci. 2020 Mar 06;21(1):10. doi: 10.1186/s12868-020-0558-9.

Following the publication of this article [1], it has been noted by the authors that an image of the same cell nuclei has been used in error twice, in Fig. 8, parts A and B. These images are redundant...

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Hooper C, Sainz-Fuertes R, Lynham S, et al. Correction to: Wnt3a induces exosome secretion from primary cultured rat microglia. BMC Neurosci. 2020;21(1):10doi: 10.1186/s12868-020-0558-9.
Hooper, C., Sainz-Fuertes, R., Lynham, S., Hye, A., Killick, R., Warley, A., Bolondi, C., Pocock, J., & Lovestone, S. (2020). Correction to: Wnt3a induces exosome secretion from primary cultured rat microglia. BMC neuroscience, 21(1), 10. https://doi.org/10.1186/s12868-020-0558-9
Hooper, Claudie, et al. "Correction to: Wnt3a induces exosome secretion from primary cultured rat microglia." BMC neuroscience vol. 21,1 (2020): 10. doi: https://doi.org/10.1186/s12868-020-0558-9
Hooper C, Sainz-Fuertes R, Lynham S, Hye A, Killick R, Warley A, Bolondi C, Pocock J, Lovestone S. Correction to: Wnt3a induces exosome secretion from primary cultured rat microglia. BMC Neurosci. 2020 Mar 06;21(1):10. doi: 10.1186/s12868-020-0558-9. PMID: 32138650; PMCID: PMC7059358.
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Overexpression of the receptor for advanced glycation end-products in the auditory cortex of rats with noise-induced hearing loss.

BMC neuroscience

Lee CH, Kim KW, Lee DH, Lee SM, Kim SY.
PMID: 34020590
BMC Neurosci. 2021 May 21;22(1):38. doi: 10.1186/s12868-021-00642-3.

BACKGROUND: The receptor for advanced glycation end-products (RAGE) is involved in neuroinflammation. This study investigated the changes in RAGE expression following noise-induced hearing loss.METHODS: Three-week-old female Sprague-Dawley rats were exposed to 115 dB SPL white noise for 4 h...

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Lee CH, Kim KW, Lee DH, et al. Overexpression of the receptor for advanced glycation end-products in the auditory cortex of rats with noise-induced hearing loss. BMC Neurosci. 2021;22(1):38doi: 10.1186/s12868-021-00642-3.
Lee, C. H., Kim, K. W., Lee, D. H., Lee, S. M., & Kim, S. Y. (2021). Overexpression of the receptor for advanced glycation end-products in the auditory cortex of rats with noise-induced hearing loss. BMC neuroscience, 22(1), 38. https://doi.org/10.1186/s12868-021-00642-3
Lee, Chang Ho, et al. "Overexpression of the receptor for advanced glycation end-products in the auditory cortex of rats with noise-induced hearing loss." BMC neuroscience vol. 22,1 (2021): 38. doi: https://doi.org/10.1186/s12868-021-00642-3
Lee CH, Kim KW, Lee DH, Lee SM, Kim SY. Overexpression of the receptor for advanced glycation end-products in the auditory cortex of rats with noise-induced hearing loss. BMC Neurosci. 2021 May 21;22(1):38. doi: 10.1186/s12868-021-00642-3. PMID: 34020590; PMCID: PMC8139161.
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